Electrophiles and Michael acceptors as Nrf2 modulators in a Caenorhabditis elegans model.

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dc.contributor.authorChong Yew, Lee
dc.date.accessioned2017-08-14T07:19:35Z
dc.date.available2017-08-14T07:19:35Z
dc.date.issued2016
dc.description.abstractAmyloid-beta (AI3) toxicity has been proposed and demonstrated to be a key contributor to the neurodegenerative process in the pathogenesis of neurodegenerative diseases such as Alzheimer's disease (AD).The mechanisms by which AI3 leads to neuronal damage and dysfunction are still not well understood. Ketoksikan Amiloid beta (A!3) didapati satu penyumbang kepada proses neurodegenerasi dalam pathogenesis penyakit Alzheimer. Mekanisme bagaimana A!3 membawa kepada kerosakan neuron masih kurang difahami.en_US
dc.identifier.urihttp://hdl.handle.net/123456789/4357
dc.subjectElectrophilesen_US
dc.subjectacceptorsen_US
dc.subjectCaenorhabditisen_US
dc.subjectNrf2 modulatorsen_US
dc.titleElectrophiles and Michael acceptors as Nrf2 modulators in a Caenorhabditis elegans model.en_US
dc.typeTechnical Reporten_US
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