Human Chromosome 4 Sequencing And Single Nucleotide Polymorphism (Snp) Analysis Of An Achondroplasia Individual

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Date
2011-02
Authors
Lee, Ling Sze
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Publisher
Universiti Sains Malaysia
Abstract
Achondroplasia is the most common cause of short-limbed dwarfism in humans, affecting 250,000 individuals worldwide. This genetic disorder results in various social and medical complications. The majority of achondroplasia cases is sporadic and result from de novo mutations. This autosomal-dominant disorder is caused by single nucleotide mutations in the gene encoding the type 3 receptor for fibroblast growth factor (FGFR3). This study focused on understanding the genetic basis of achondroplasia by identifying SNPs from flow-sorted human chromosomes of an achondroplasia volunteer of Asian origin. Chromosome staining and the bivariate flow karyotyping of human chromosomes were successfully optimized. Whole Genome Amplification (WGA) was carried out to generate high-throughput sequencing data. Thorough analysis of the sequence data and SNPs was unable to identify any known mutations of achondroplasia and hypochondroplasia. Thus, it indicates that the classical achondroplasia indicator gene, fgfr3, may not be the only indicator in this particular case.
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Keywords
Human Chromosome , Single Nucleotide Polymorphism (Snp)
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